The pathophysiology and treatment of sepsis.
By R. Hotchkiss, I. Karl. New England Journal of Medicine, 2003
4,150 citations recorded by the source corpus.
Subfields: Epidemiology, Immunology, Nutrition and Dietetics
View Paper: https://www.semanticscholar.org/paper/29e895e021d7ba5a093046698656d3d5e9a66833
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Citations in the corpus
- Sepsis and the heart. (2007, neutral): “(2) these acute changes in end-diastolic volume index and”
- Apoptosis and caspases regulate death and inflammation in sepsis (2006, background): “The magnitude and duration of the initial hyper-inflammatory state is determined by several factors, including the amount and virulence of the invading microorganisms, the genetic predisposition of the host and coexistent medical conditions of the patients (co-morbiditie”
- Persistent inflammation and immunosuppression: A common syndrome and new horizon for surgical intensive care (2012, background): “It acts as an antiapoptotic cytokine and augments interferon F expression, which may help reverse monocyte dysfunction in sepsis and, therefore, is an intriguing immunostimulatory cytokine.(72) In a murine model of polymicrobial sepsis, Inoue et al.”