Apoptosis and caspases regulate death and inflammation in sepsis

By R. Hotchkiss, D. Nicholson. Nature reviews. Immunology, 2006

815 citations recorded by the source corpus.

Subfields: Immunology, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/64d932c2a256ef4b0e1c450fcc742aa774ff4e82

How researchers see this paper

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In Immunology

1. R. Bucala
2. D. Remick

In Molecular Biology

1. D. Remick

Citations in the corpus

• Cytokines in Sepsis: Potent Immunoregulators and Potential Therapeutic Targets—An Updated View (2013, background): “More recently, it has been proposed that the early phase of hyperinflammation is followed or overlapped by a prolonged state of immunosuppression [26–28], referred to as sepsisinduced immunoparalysis [29].”
• The Pathogenesis of Sepsis (2011, background): “, caspase-3 and caspase-8) and CD95, and most recently the use of noncaspase protease inhibitors (43).”