TICAM-1, an adaptor molecule that participates in Toll-like receptor 3–mediated interferon-β induction

By H. Oshiumi, Misako Matsumoto, Kenji Funami, T. Akazawa, Tsukasa Seya. Nature Immunology, 2003

1,089 citations recorded by the source corpus.

Subfields: Immunology, Microbiology

View Paper: https://www.semanticscholar.org/paper/254eba7467e9da33e95c644ee9190c8fab0ad789

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Immunology

1. Shizuo Akira
2. Taro Kawai
3. S. Ghosh
4. Satoshi Uematsu
5. M. Hayden
6. K. Takeda
7. A. West
8. R. Medzhitov

In Microbiology

1. K. Takeda
2. Shizuo Akira
3. Taro Kawai
4. Satoshi Uematsu
5. S. Ghosh
6. R. Medzhitov

Citations in the corpus

• Pathogen Recognition and Inflammatory Signaling in Innate Immune Defenses (2009, neutral): “Thus, molecular signaling mechanisms within the TRIF-dependent pathway illustrate how selective binding of different molecules, i.e., either TRAF3 or TRAF6, results in recruitment and downstream activation of TBK1-IRF versus TAK1-IKK-NF- B, respectively (Fig.”
• Toll-like receptors and innate immunity (2006, background): “However, cells doubly deficient in TRAF6 and MyD88 still partially activated NF-κB in response to LPS, suggesting that TRIF activates NF-κB through both TRAF6-dependent and TRAF6-independent pathways in TLR4 signaling [65, 66].”