Multiple Sclerosis
By Ralph Benedict1, Julie Bobholz2. Journal - Michigan State Medical Society, 2007
11,454 citations recorded by the source corpus.
Subfields: Pathology and Forensic Medicine
View Paper: https://www.semanticscholar.org/paper/22d31d11e02ba907760be23c100835f14e7cc338
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In Pathology and Forensic Medicine
- Hans Lassmann
- Wolfgang Brück
- C. Lucchinetti
- Robin J. M. Franklin
- Sandra Vukusic
- Christian Confavreux
- Stephen L. Hauser
- K. Munger
Citations in the corpus
- The Immunopathology of Multiple Sclerosis: An Overview (2007, background): “Furthermore, current anti-inflammatory, immunomodulatory or immunosuppressive treatments are at least partly effective in the early stages of the disease but are of limited benefit when patients have entered the progressive phase (68).”
- Chronic cerebrospinal venous insufficiency in patients with multiple sclerosis (2008, background): “Multiple sclerosis (MS) is an inflammatory demyelinating disease of the central nervous system (CNS) of unknown pathogenesis.(1) 2 MR venography and postmortem studies(7) have demonstrated a topographic correspondence between MS plaques and cerebral venous system.”
- Clinical and Pathological Insights into the Dynamic Nature of the White Matter Multiple Sclerosis Plaque (2015, background): “Multiple Sclerosis (MS) is the most common cause of nontraumatic disability in young adults.(1) MS classically has been described as a chronic, inflammatory, demyelinating disease of the central nervous system (CNS) with the focal white matter lesion, the “plaque,” as its hallmark pathology.”
- BRAIN REGENERATION IN PHYSIOLOGY AND PATHOLOGY: THE IMMUNE SIGNATURE DRIVING THERAPEUTIC PLASTICITY OF NEURAL STEM CELLS (2011, background): “This leads to highly heterogeneous, chronic inflammatory, demyelinating multifocal CNS lesions (44, 60, 243).”
- Progesterone receptors: form and function in brain. (2008, neutral): “It is interesting to note that women have a greater risk of developing the demylinating disease multiple sclerosis with a frequent onset at menopause [207].”