Lethal influenza infection in the absence of the natural killer cell receptor gene Ncr1
By R. Gazit, Raizy Gruda, Moran Elboim, T. Arnon, Gil Katz, H. Achdout, J. Hanna, U. Qimron, Guy Landau, E. Greenbaum and 3 more. Nature Immunology, 2006
599 citations recorded by the source corpus.
Subfields: Epidemiology, Immunology
View Paper: https://www.semanticscholar.org/paper/203c27902de9f78e935f0bf55edef161045cd671
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In Immunology
- Chao Yang
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Citations in the corpus
- Microbial flora drives interleukin 22 production in intestinal NKp46+ cells that provide innate mucosal immune defense. (2008, background): “Identification of Phenotypically Diverse NK Cell Subsets in the Murine Intestinal Tract NK cells from several species have been shown to specifically express the natural cytotoxicity receptor NKp46 (encoded by the Ncr1 locus) (Biassoni et al., 1999; Gazit et al., 2006; Sivori et al., 1997; Walzer et”
- The Airway Epithelium: Soldier in the Fight against Respiratory Viruses (2011, background): “A study demonstrating that influenza virus infection was lethal in mice lacking the NK cell-activating receptor NCR1 suggests that NK cells are indeed crucial for protection against respiratory virus infections (78).”
- Modulation of natural killer cell activity by viruses (2010, unknown): “NCR1 knock-out mice are more sensitive to influenza virus than their wild-type counterparts [45].”
- Natural Killer Cells: Development, Maturation, and Clinical Utilization (2018, background): “1 (+) NKG2A/C (+) NCR1 (+) CD43 (+) CD62L (+) CD226 (+) CD122 (+) CD27 (+) CD244 (+) NKG2D (+) NK1.”