Bone marrow-derived progenitor cells in pulmonary fibrosis.

By N. Hashimoto, Hong Jin, Tianju Liu, S. Chensue, S. Phan. Journal of Clinical Investigation, 2004

755 citations recorded by the source corpus.

Subfields: Genetics, Pulmonary and Respiratory Medicine

View Paper: https://www.semanticscholar.org/paper/1ef3cd63b3a39981ff77cc5c4f36ab03440afc63

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Genetics

1. Brigham C. Willis
2. B. Hinz
3. Z. Borok

In Pulmonary and Respiratory Medicine

1. B. Hogan
2. J. Rock
3. P. Noble
4. C. Feghali-Bostwick
5. Brigham C. Willis
6. B. Hinz
7. Ryan T. Kendall
8. Z. Borok

Citations in the corpus

• Formation and function of the myofibroblast during tissue repair. (2007, background): “, 2003) and (2) from transgenic green fluorescent mice to irradiated wild-type animals (Hashimoto et al., 2004).”
• Induction of epithelial-mesenchymal transition in alveolar epithelial cells by transforming growth factor-beta1: potential role in idiopathic pulmonary fibrosis. (2005, background): “1,14 Although the morphology of epithelial cells in samples of lung tissue from patients with IPF is highly abnormal, with significant alterations in morphology and phenotypic marker expression, 4,37 the possibility that AECs may participate in the development of pulmonary fibrosis through…”
• Multiple stromal populations contribute to pulmonary fibrosis without evidence for epithelial to mesenchymal transition (2011, background): “A second proposed source of fibrotic cells is circulating bone marrow-derived fibrocytes (8, 9).”
• Circulating fibrocytes traffic to the lungs in response to CXCL12 and mediate fibrosis. (2004, positive): “In addition, recent evidence has indicated that in vitro bone marrow–derived lung CXCR4+ cells can migrate in response to CXCL12 (23).”
• Fibroblasts in fibrosis: novel roles and mediators (2014, background): “…into chimeric normal mice appear to be a unique population of cells that do not express αSMA and fail to differentiate into myofibroblasts when extracted and stimulated ex vivo in culture (Hashimoto et al., 2004; Kisseleva et al., 2006; Barisic-Dujmovic et al., 2010; Rock et al., 2011).”