[DNA methylation and cancer].

By Li Yu. Zhonghua nei ke za zhi, 2005

2,054 citations recorded by the source corpus.

View Paper: https://www.semanticscholar.org/paper/1d5801222ef4d347d22047649b72d00482e53cea

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Citations in the corpus

Posttranslational Protein Modification in Archaea (2005, background): “Although methylation of nucleic acids is well known, in part due to a role in disease states such as cancer (86, 256, 424), a wide variety of proteins have also been reported to experience posttranslational methylation.”
Dynamics of DNA damage response proteins at DNA breaks: a focus on protein modifications. (2011, background): “…acts as a physical barrier to the detection and repair of DNA lesions, it is also a dynamic structure that can be modulated by DNA methylation (Kulis and Esteller 2010), incorporation of histone variants (Bernstein and Hake 2006), histone post-translational modifications (Kouzarides 2007),…”
Dynamic DNA methylation across diverse human cell lines and tissues (2013, background): “…Genome Sciences and Policy, Duke University, Durham, North Carolina 27708, USA As studies of DNA methylation increase in scope, it has become evident that methylation has a complex relationship with gene expression, plays an important role in defining cell types, and is disrupted in many diseases.”
Epigenome-Wide Association Studies for common human diseases (2011, background): “However, there is now increasing interest in exploring how non-genetic variation, including epigenetic factors, could influence complex disease aetiolog”
Oxidative stress, inflammation, and cancer: how are they linked? (2010, background): “This leads to promoter silencing of several genes, such as adenomatous polyposis coli, cyclindependent kinase inhibitor-2, breast cancer susceptibility gene 1, retinoblastoma protein, murine double minute 2 (MDM2), and the DNA mismatch repair gene, human mutL homolog 1 [96,97].”
Molecular targets of dietary agents for prevention and therapy of cancer. (2006, background): “Hypomethylation is another methylation defect that is observed in a wide variety of cancers including hepatocellular cancers, cervical cancer, prostate tumors and hematologic malignancies (for references see [222]).”