Estrogen receptors: how do they signal and what are their targets.
By N. Heldring, A. Pike, S. Andersson, J. Matthews, G. Cheng, Johan Hartman, M. Tujague, A. Ström, E. Treuter, M. Warner and 1 more. Physiological Reviews, 2007
1,835 citations recorded by the source corpus.
Subfields: Genetics, Oncology, Toxicology
View Paper: https://www.semanticscholar.org/paper/193d24c96129f276f6af2fb6b156cfdcbc19259a
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In Genetics
In Oncology
Citations in the corpus
- Endocrine Disrupting Chemicals and Disease Susceptibility (2011, background): “This contrasts with the estrogen receptors (ERα and ERβ), which have been extensively studied [7].”
- The different roles of ER subtypes in cancer biology and therapy (2011, background): “Oestrogens mediate their effects in target tissues through two members of the nuclear receptor superfamily, oestrogen receptor-α (ERα) and ER”
- Estrogen receptors regulate innate immune cells and signaling pathways (2015, background): “This is consistent with prior work showing that MISS pathways can lead to histone modifications and altered chromatin structure near estrogen-regulated genes [1, 22].”
- Exactly the same but different: promiscuity and diversity in the molecular mechanisms of action of the aryl hydrocarbon (dioxin) receptor. (2011, background): “The ER exists as two isoforms (ERa and ERb) that are localized primarily in the nucleus in their unliganded state and are found complexed with two hsp90 chaperone proteins and several other protein factors (Beischlag et al., 2008; Heldring et al., 2007).”