How do plants feel the heat?

By R. Mittler, Andrija Finka, P. Goloubinoff. TIBS -Trends in Biochemical Sciences. Regular ed, 2012

922 citations recorded by the source corpus.

Subfields: Molecular Biology, Plant Science

View Paper: https://www.semanticscholar.org/paper/19181a36851dec53cbf76e232154e8aab829568f

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Citations in the corpus

Reactive oxygen species, abiotic stress and stress combination. (2017, background): “In contrast, signaling ROS is generated as a response to stress perception by stress sensors (e.g. cyclic nucleotide-gated channels activated by heat stress; Mittler et al., 2012) and is mediated by calcium- and/or phosphorylation-derived activation of NADPH oxidases (RBOH) at the plasma membrane (P”
Abiotic and biotic stress combinations. (2014, background): “…inhibited as a result of the low temperature or insufficient availability of CO2, and the high photosynthetic energy absorbed by the plant (as a result of the high light intensities) enhances oxygen reduction and thus reactive oxygen species (ROS) production (Mittler, 2002; Mittler et al., 2004).”
Food crops face rising temperatures: An overview of responses, adaptive mechanisms, and approaches to improve heat tolerance (2016, background): “Heat stress leads to the activation of unfolded proteins (UPR) present in endoplasmic reticulum (ER-UPR) and cytosol (Cyt-UPR) (Deng, Srivastava, & Howell, 2013; Mittler et al., 2012).”
Complexity of the Alternative Splicing Landscape in Plants[C][W][OPEN] (2013, background): “…changes in messengers such as calcium and reactive oxygen species, which are known regulate transcription of numerous genes (Reddy et al., 2011; Mittler et al., 2012; Smékalová et al., 2013), may also regulate AS through protein kinases and protein phosphatases as the activity of many splicing…”
The effect of high temperature stress on male and female reproduction in plants (2015, background): “Physical changes in membranes, cellular calcium and ROS, to include H2O2, stimulate a network of signal transduction pathways, heat shock proteins, and transcriptional regulators to re-establish cellular homeostasis (Saidi et al., 2011; Mittler et al., 2012; Fragkostefanakis et al., 2014).”