Prospective study of caffeine consumption and risk of Parkinson's disease in men and women

By A. Ascherio, Shumin M. Zhang, Miguel A. Hern'an, Ichiro Kawachi, Graham A Colditz, F. Speizer, W. Willett. Annals of Neurology, 2001

677 citations recorded by the source corpus.

Subfields: Neurology, Nutrition and Dietetics, Pharmacology

View Paper: https://www.semanticscholar.org/paper/15657189d0d41afbabb8bef8b2237d067b73d96e

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In Neurology

1. P. Jenner
2. A. Schapira
3. D. Dexter

Citations in the corpus

• Epidemiology and etiology of Parkinson’s disease: a review of the evidence (2011, background): “In the Health Professionals Follow-up Study [478], there was an inverse association between tea intake and PD in men who did not drink coffee (RR 0.”
• Etiology and pathogenesis of Parkinson's disease (2011, background): “The evidence for cigarette smoking and caffeine intake in reducing risk appears clear, but there is still uncertainty over the role of others, for example, exercise, anti-inflammatories, antihypertensives (most notably calcium antagonists), and antilipidaemics.(20,21) Although many specific examples”
• A meta‐analysis of coffee drinking, cigarette smoking, and the risk of Parkinson's disease (2002, background): “Arguments against the reverse causation hypothesis are that it would imply a very early onset of PD, that animal experiments have found a neuroprotective effect of caffeine in mice exposed to compounds that cause parkinsonism,(63) that caffeine from noncoffee sources (eg, tea, cola beverages, chocol”