The antioxidant paradox.

By Barry Halliwell. The Lancet, 2000

700 citations recorded by the source corpus.

Subfields: Biochemistry, Molecular Biology, Nutrition and Dietetics

View Paper: https://www.semanticscholar.org/paper/139dad0b1f32c39a2715a35e05d95db2817c920f

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Citations in the corpus

A systematic screening of total antioxidants in dietary plants. (2002, result): “This would be in accordance with the observation that brussels sprouts, onion and tomatoes (27–30) but not single antioxidants such as -carotene, vitamins E, vitamin C or coenzyme Q10 are able to reduce the excretion of biomarkers for free radical DNA damage in urine or lymphocytes in humans (18,21,”
Measuring reactive species and oxidative damage in vivo and in cell culture: how should you do it and what do the results mean? (2004, background): “The advantages and artifacts of measuring 8OHdG in cellular DNA have been recently reviewed (Halliwell, 2000c; 2002b; Collins et al., 2004), so they need not be reiterated here.”
Chronic Conditions, Functional Limitations, and Special Health Care Needs of School-Aged Children Born With Extremely Low-Birth-Weight in the 1990s (2005, positive): “In particular, we were interested in whether levels of oxidative stress may modify the effect of vitamin E.(50) We did not have a direct measure of oxidative stress; however, smoking and diseases such as hypertension, hyperlipidemia, and diabetes are associated with increased production of reactive ”
Reactive Species and Antioxidants. Redox Biology Is a Fundamental Theme of Aerobic Life (2006, background): “Simply because the human body regulates the ROS/antioxidant balance so carefully that feeding antioxidants does not disturb it much, and oxidative damage does not decrease (Halliwell, 1999, 2000).”
Invited Review: Oxidation of Biological Systems: Oxidative Stress Phenomena, Antioxidants, Redox Reactions, and Methods for Their Quantification (2002, background): “In these latter stages the cellular environment becomes highly oxidized (95).”