Impaired Myofibrillar Energetics and Oxidative Injury During Human Atrial Fibrillation

By M. Mihm, F. Yu, C. Carnes, P. Reiser, P. McCarthy, D. V. Van Wagoner, J. Bauer. Circulation, 2001

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Arrhythmogenic ion-channel remodeling in the heart: heart failure, myocardial infarction, and atrial fibrillation. (2007, background): “There is evidence for oxidative injury in atrial tissues from AF patients (197) and atrial tachypaced-remodeled dogs (48).”
Oxidative stress in the placenta (2004, background): “…nitrated proteins have been identified in- cluding myofibrillar creatine kinase (Mihm et al. 2001a), PGI2 synthase (Zou et al. 1999), and heart succinylCo-A:3-oxoacid CoA transferase (Turko et al. 2001), and structural proteins such as myosin heavy chain, a-actinin, and desmin (Mihm et al. 2001b).”
Protein carbonyl groups as biomarkers of oxidative stress. (2003, background): “Alzheimer’s disease [46,54–57,60,61,103] Atrial fibrillation [104] Bronchopulmonary dysplasia [58] Chronic renal failure/uremia [44,59] Duchenne and Becker muscular dystrophy [105] Helicobacter pylori infection and inflammation [106] Parkinson’s disease [107] Psoriasis [108] I.”