Controversies in ventricular remodelling.

By L. Opie, P. Commerford, B. J. Gersh, M. Pfeffer. The Lancet, 2006

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Left ventricular remodeling in heart failure: current concepts in clinical significance and assessment. (2011, background): “Although riginally described after myocardial infarction MI), ventricular remodeling develops in reponse to a variety of forms of myocardial njury and increased wall stress (4,5).”
Myocardial remodeling after infarction: the role of myofibroblasts (2010, background): “In addition to interstitial alterations in the noninjured areas, replacement fibrosis, although it initially supports ventricular morphology after MI, can contribute to geometric changes and inevitable functional deterioration over time”
Pathological Ventricular Remodeling: Mechanisms: Part 1 of 2 (2013, background): “to minimizing infarct size and maintaining ventricular performance.(45) Significant advances in our understanding of”
Inflammatory responses and inflammation-associated diseases in organs (2015, background): “Necrotic cardiac cells initiate an inflammatory cascade to clear dead cells and debris from the infarct [95, 96].”
The role of TGF-β Signaling in Myocardial Infarction and Cardiac Remodeling (2007, background): “Pathologic remodeling occurs in three major patterns: a) concentric remodeling when pressure overload causes growth in cardiomyocyte thickness, b) eccentric remodeling resulting from a volume load that produces cardiomyocyte lengthening and c) post-infarction remodeling, which involves a combined pr”