Left ventricular remodeling after myocardial infarction: pathophysiology and therapy.

By M. J. Sutton, N. Sharpe. Circulation, 2000

1,851 citations recorded by the source corpus.

View Paper: https://www.semanticscholar.org/paper/09ab61865a2457ea1878510ef71e3245015aea0f

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Citations in the corpus

Cardiomyocyte A Disintegrin And Metalloproteinase 17 (ADAM17) Is Essential in Post–Myocardial Infarction Repair by Regulating Angiogenesis (2015, background): “Angiogenesis has the potential to rescue the ischemic myocardium at early stages after MI and can prevent the transition to heart failure.(11,12) ADAM17, particularly in endothelial cells, has been shown to promote angiogenesis through different mechanisms.”
Cardiac fibrosis in myocardial infarction—from repair and remodeling to regeneration (2016, background): “The remodeling process includes thickening (hypertrophy) and stiffening (fibrosis) of the left ventricular wall (Sutton and Sharpe 2000).”
Myocardial matrix remodeling and the matrix metalloproteinases: influence on cardiac form and function. (2007, background): “In these instances, structural changes occur within the myocardial wall that result in changes in LV geometry, a process termed myocardial remodeling (7, 62, 75, 76, 102, 135, 209, 310, 335, 426, 429, 487, 494, 495).”
Atlas-Based Quantification of Cardiac Remodeling Due to Myocardial Infarction (2014, background): “In patients with cardiovascular disease, certain changes in heart shape over time, known as remodeling, are indicative of worse prognostic outcome [6].”
Assessment of Myocardial Fibrosis with Cardiac Magnetic Resonance (2011, background): “This replacement or scarring fibrosis corresponds to the replacement of myocytes after cell damage or necrosis by plexiform fibrosis, mainly type I collagen (37).”
Orthogonal decomposition of left ventricular remodeling in myocardial infarction (2017, background): “Relative LV wall thickness [1] and apical conicity [7] are also important indices of adverse remodeling after myocardial infarction.”
The Biological Basis for Cardiac Repair After Myocardial Infarction: From Inflammation to Fibrosis. (2016, background): “91 Adverse left ventricular (LV) remodeling after myocardial infarction (MI) constitutes the structural basis for ischemic heart failure (HF) and comprised complex short- and long-term changes in LV size, shape, function, and cellular and molecular composition.(1,2) Although multiple pathophysiologi”
Targeted Imaging of the Spatial and Temporal Variation of Matrix Metalloproteinase Activity in Porcine Model of Post-Infarct Remodeling: Relationship to Myocardial Dysfunction (2011, methodology): “Accordingly, the aims of the current study were 2-fold: (1) validate an MMP-targeted radiotracer approach for in vivo evaluation of MMP activation after MI, comparing radiotracer-derived indices of regional MMP activation with quantitative MMP zymography, and (2) use hybrid SPECT/CT imaging of MMP a”