Retinoids in embryonal development.
By S. Ross, P. McCaffery, U. C. Dräger, L. M. Luca. Physiological Reviews, 2000
893 citations recorded by the source corpus.
Subfields: Cellular and Molecular Neuroscience, Genetics, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/0761725c706df90b5ad45dfcdba36f2e2d56181a
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In Genetics
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Citations in the corpus
- Gene expression regulation by retinoic acid Published, JLR Papers in Press, August 16, 2002. DOI 10.1194/jlr.R100015-JLR200 (2002, background): “Ross, S. A., P. J. McCaffery, U. C. Drager, and L. M. De Luca.”
- Nuclear retinoid receptors and the transcription of retinoid-target genes. (2004, background): “Vitamin A and its active derivatives referred to as retinoids are non-steroid hormones which play a critical role in the development and homeostasis of virtually every vertebrate tissues through their regulatory effects on cell differentiation, proliferation and apoptosis (Ross et al., 2000; Altucci”
- Effect of dietary vitamin A on Senegalese sole (Solea senegalensis) skeletogenesis and larval quality (2009, background): “, 2007) due to the morphogenetic action of this 92 nutrient, which is known to have teratogenic effects in vertebrates at inappropriate 93 dietary levels (Ross et al., 2000).”
- Nutritional components affecting skeletal development in fish larvae (2003, background): “The teratogenic effect of retinoic acid, a compound related to vitamin A, is now well documented in vertebrates (Ross et al., 2000).”
- Skeletal anomalies in reared European fish larvae and juveniles. Part 2: main typologies, occurrences and causative factors (2013, background): “The term vitamin A is used to designate any compound possessing the biological activity of retinol (alcohol form), whereas the term retinoids applies to a group of morphogenetic nutrients that include all compounds that possess the same biological activity as retinol (Ross et al. 2000).”
- Variations on development and stress defences in Solea senegalensis larvae fed on live and microencapsulated diets (2006, background): “VA, either in excess or in deficiency, has been reported to be teratogenic in vertebrates (Ross et al., 2000) and in fish larvae (Takeuchi et al.”