Dysbiosis Contributes to Arthritis Development via Activation of Autoreactive T Cells in the Intestine

By Y. Maeda, T. Kurakawa, E. Umemoto, D. Motooka, Yoshinaga Ito, Kazuyoshi Gotoh, K. Hirota, M. Matsushita, Yoki Furuta, M. Narazaki and 8 more. Arthritis & Rheumatology, 2016

598 citations recorded by the source corpus.

Subfields: Infectious Diseases, Molecular Biology, Rheumatology

View Paper: https://www.semanticscholar.org/paper/057c5d205cc61ba03b7d039945f86b6b7bf307b9

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Infectious Diseases

1. Eran Elinav

In Molecular Biology

1. D. Zheng
2. S. Gaffen
3. Eran Elinav

In Rheumatology

1. S. Gaffen

Citations in the corpus

• Interaction between microbiota and immunity in health and disease (2020, background): “Moreover, genetically susceptible mice colonized with dysbiotic microbiota from RA patients show an enhanced Th17 type response.(169) Similarly, inoculation of SFB into GF mice is sufficient to induce Th17 activation and to instigate autoimmune arthritis.”
• The IL-17 family of cytokines in health and disease (2019, background): “, 2013), and have been demonstrated to contribute to arthritis susceptibility in mice (Maeda et al., 2016; Wu et al., 2010).”