Modeling the excitability of mammalian nerve fibers: influence of afterpotentials on the recovery cycle.

By Cameron C. McIntyre, Andrew G. Richardson, W. Grill. Journal of Neurophysiology, 2002

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Patient-specific analysis of the volume of tissue activated during deep brain stimulation (2007, background): “7 μm) myelinated axons (McIntyre et al., 2002; Butson and McIntyre, 2006), and may not be representative of the response of other neuron types surrounding the electrode (local projection neurons, local interneurons, afferent inputs, etc.”
Cellular effects of deep brain stimulation: model-based analysis of activation and inhibition. (2004, background): “distribution and was able to capture nearly every experimentally recorded firing property of myelinated axons [McIntyre et al., 2002].”
Mechanisms of axon ensheathment and myelin growth (2005, result): “Decreased internodal lengths did reduce rates of conduction in accordance with predictions from a recent, rather sophisticated mathematical mode”
Computational analysis of subthalamic nucleus and lenticular fasciculus activation during therapeutic deep brain stimulation. (2006, methodology): “The axon of the STN neuron and the GPi and CST axons were based on the myelinated axon model originally described by McIntyre et al. (2002).”