Type 2 (non-insulin-dependent) diabetes mellitus, hypertension and hyperlipidaemia (syndrome X): relation to reduced fetal growth
By D. Barker, C. Hales, C. Fall, C. Osmond, K. Phipps, P. Clark. Diabetologia, 2004
2,181 citations recorded by the source corpus.
Subfields: Obstetrics and Gynecology, Pediatrics, Perinatology and Child Health
View Paper: https://www.semanticscholar.org/paper/01ddbda14442f7d453a9e386126d26e334ec16d3
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In Obstetrics and Gynecology
In Pediatrics, Perinatology and Child Health
Citations in the corpus
- Racial and Ethnic Disparities in Birth Outcomes: A Life-Course Perspective (2003, background): “factors on lifelong chances of developing coronary heart disease (46), diabetes mellitus (47), and hypertension (48).”
- Fetal origins of obesity. (2003, background): “A first potential pathway suggests that inadequate maternal nutrient intake and low availability of protein cause permanent changes in pancreatic vascularity, structure, and function (93,96).”
- Birth weight and subsequent risk of type 2 diabetes: a meta-analysis. (2007, background): “(1) published highly influential findings indicating a relation between low birth weight and increased risk of developing symptoms of the metabolic syndrome.”
- Developmental origins of the metabolic syndrome: prediction, plasticity, and programming. (2005, neutral): “Insulin resistance (with or without glucose intolerance), raised blood pressure, atherogenic dyslipidemia [elevated triglyceride, small low-density lipoprotein (LDL) particles, low high-density lipoprotein (HDL) cholesterol], central or abdominal obesity, and prothrombotic and proinflammatory states”
- Prenatal glucocorticoids and long-term programming. (2004, background): “…initially in the UK and then world-wide, have revealed an association between lower birth weight and the subsequent development of the common cardiovascular and metabolic disorders of adult life, notably hypertension, insulin resistance, type 2 diabetes and cardiovascular disease deaths (2, 26–34).”
- The stunting syndrome in developing countries (2014, background): “…Origins of Health and Disease’ hypothesis proposes that nutritional deprivation during fetal or infant life 252–254 triggers permanent epigenetic changes in metabolism (e.g. of lipids 253 and glucose 252 ) and organ anatomy and function (e.g. of blood vessels, liver and kidney 255–257 ).”
- Endocrine regulation of human fetal growth: the role of the mother, placenta, and fetus. (2006, background): “An inverse correlation between placental weight and the length to head circumference ratio suggested the possibility that changes in the fetal circulation, such as diversion of blood flow to the brain at the expense of other parts of the body, may lead to permanent alterations in blood vessel develo”