Prenatal glucocorticoids and long-term programming.

By J. Seckl. European Journal of Endocrinology, 2004

872 citations recorded by the source corpus.

Subfields: Nutrition and Dietetics, Pediatrics, Perinatology and Child Health, Pulmonary and Respiratory Medicine

View Paper: https://www.semanticscholar.org/paper/78a8ad8913133b00eb0ad730785c9a326474a549

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In Nutrition and Dietetics

In Pediatrics, Perinatology and Child Health

In Pulmonary and Respiratory Medicine

Citations in the corpus

Maternal adversity and ecological stressors in natural populations: the role of stress axis programming in individuals, with implications for populations and communities (2013, background): “The sensitivity of the feedback response is due to the level of GCs and the number of GC and mineralcorticoid receptors (GR and MR, respectively) in the brain and GR in the body (de Kloet et al. 1998; Wingfield & Sapolsky 2003; Seckl 2004).”
Prenatal Stress, Glucocorticoids and the Programming of Adult Disease (2009, background): “Numerous studies across a wide range of species have shown that prenatal treatment with glucocorticoids reduces birth weight and that these offspring are at increased risk of cardio-metabolic disease, hypothalamic–pituitary–adrenal (HPA) axis perturbations and affective disorders in later life (Seck”
Transgenerational effects of posttraumatic stress disorder in babies of mothers exposed to the World Trade Center attacks during pregnancy. (2005, background): “testing, other potential hypothesized mechanisms, related to early social regulation (13), glucocorticoid programing in utero (6), and/or shared underlying genetic susceptibility (14) are more relevant to the cortisol alterations observed.”
Prenatal developmental origins of behavior and mental health: The influence of maternal stress in pregnancy. (2017, background): “…system (ANS), neuroendocrine (hypothalamic-pituitary-adrenal (HPA) axis), cardiovascular, and immune systems (Bale, 2015; Bale et al., 2010; Griffiths and Hunter, 2014; Harris and Seckl, 2011; Meaney et al., 2007; Räikkönen et al., 2011; Seckl, 2004; Stroud et al., 2016; Stroud et al., 2014).”
Measuring stress in wildlife: techniques for quantifying glucocorticoids (2011, background): “This negative feedback loop regulates the normal circadian rhythm of GC release (De Kloet et al. 1998; Wingfield and Sapolsky 2003; Seckl 2004).”
Congenital adrenal hyperplasia due to steroid 21-hydroxylase deficiency: an Endocrine Society clinical practice guideline. (2010, positive): “Because newborn birth weight correlates with adult incidences of ischemic heart disease and hypertension (84,85) and because moderately low-dose dexamethasone (100 μg/kg) reduced birth weight and increased blood pressure in rats (86), concern was raised about the effects of prenatal dexamethasone tr”