Tadashi Yoshida

Molecular Biology · with Delafontaine · 2020

A researcher in the Science corpus: 1 paper; trusts 2 researchers and is trusted by 0 researchers here, as inferred from the citation graph.

Subfields

Whom Tadashi Yoshida trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. M. Sandri
  2. N. Mizushima

Papers from Tadashi Yoshida's perspective

Ordered by how strongly the researchers Tadashi Yoshida trusts endorse each paper. Same corpus, a different order for every researcher.

  1. IKKbeta/NF-kappaB activation causes severe muscle wasting in mice. (Cell)
  2. Identification of Ubiquitin Ligases Required for Skeletal Muscle Atrophy (Science)
  3. FoxO3 controls autophagy in skeletal muscle in vivo. (Cell Metabolism)
  4. Akt/mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivo (Nature Cell Biology)
  5. IGF-I stimulates muscle growth by suppressing protein breakdown and expression of atrophy-related ubiquitin ligases, atrogin-1 and MuRF1. (American Journal of Physiology. Endocrinology and Metabolism)
  6. Mediation of IGF-1-induced skeletal myotube hypertrophy by PI(3)K/Akt/mTOR and PI(3)K/Akt/GSK3 pathways (Nature Cell Biology)
  7. FoxO3 coordinately activates protein degradation by the autophagic/lysosomal and proteasomal pathways in atrophying muscle cells. (Cell Metabolism)
  8. The E3 Ligase MuRF1 degrades myosin heavy chain protein in dexamethasone-treated skeletal muscle. (Cell Metabolism)
  9. Myostatin reduces Akt/TORC1/p70S6K signaling, inhibiting myoblast differentiation and myotube size. (American Journal of Physiology - Cell Physiology)
  10. Autophagy is required to maintain muscle mass. (Cell Metabolism)

Their papers