D. Altomare
Molecular Biology · 2005
A researcher in the Science corpus: 1 paper; trusts 5 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 23 citations into it
Whom D. Altomare trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
- K. Inoki
- David Carling
- Julian Downward
- A. Bellacosa
- J. Testa
- P. Pandolfi
- D. Hanahan
- Jiyong Liang
- William Y. Kim
- I. Shiojima
Who trusts D. Altomare overall
Papers from D. Altomare's perspective
Ordered by how strongly the researchers D. Altomare trusts endorse each paper. Same corpus, a different order for every researcher.
- Enhanced sensitivity of PTEN-deficient tumors to inhibition of FRAP/mTOR (Proceedings of the National Academy of Sciences of the United States of America)
- TSC2 mediates cellular energy response to control cell growth and survival. (Cell)
- mTOR inhibition reverses Akt-dependent prostate intraepithelial neoplasia through regulation of apoptotic and HIF-1-dependent pathways (Nature Medicine)
- High Frequency of Mutations of the PIK3CA Gene in Human Cancers (Science)
- An inhibitor of mTOR reduces neoplasia and normalizes p70/S6 kinase activity in Pten+/− mice (Proceedings of the National Academy of Sciences of the United States of America)
- PTEN: One Gene, Many Syndromes (Human Mutation)
- Dysregulation of the TSC-mTOR pathway in human disease (Nature Genetics)
- Activation of AKT kinases in cancer: implications for therapeutic targeting. (Advances in Cancer Research)
- The biology and clinical relevance of the PTEN tumor suppressor pathway. (Journal of Clinical Oncology)
- PIK3CA mutations correlate with hormone receptors, node metastasis, and ERBB2, and are mutually exclusive with PTEN loss in human breast carcinoma. (Cancer Research)
Their papers
- Perturbations of the AKT signaling pathway in human cancer (2005) — 1,360 citations