C. Hensley

Cancer Research · Cell · 2013–2017

A researcher in the Science corpus: 4 papers; trusts 2 researchers and is trusted by 3 researchers here, as inferred from the citation graph.

Subfields

Whom C. Hensley trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. C. V. Dang
  2. Ralph J. Deberardinis
  3. Gerhard Christofori
  4. A. Schulze
  5. David R. Wise

Who trusts C. Hensley overall

  1. S. Weinberg
  2. Ralph J. Deberardinis
  3. Navdeep S. Chandel
  4. Kirsten L. Bryant
  5. Laura A. Sena
  6. Michael D. Buck
  7. J. Mancias
  8. Zachary E. Stine
  9. E. Panieri
  10. B. Altman

Papers from C. Hensley's perspective

Ordered by how strongly the researchers C. Hensley trusts endorse each paper. Same corpus, a different order for every researcher.

  1. c-Myc suppression of miR-23 enhances mitochondrial glutaminase and glutamine metabolism (Nature)
  2. Myc regulates a transcriptional program that stimulates mitochondrial glutaminolysis and leads to glutamine addiction (Proceedings of the National Academy of Sciences of the United States of America)
  3. Understanding the Warburg Effect: The Metabolic Requirements of Cell Proliferation (Science)
  4. Phosphate-activated glutaminase (GLS2), a p53-inducible regulator of glutamine metabolism and reactive oxygen species (Proceedings of the National Academy of Sciences of the United States of America)
  5. The Key Role of Anaplerosis and Cataplerosis for Citric Acid Cycle Function* (Journal of Biological Chemistry)
  6. Acetate is a Bioenergetic Substrate for Human Glioblastoma and Brain Metastases (Cell)
  7. Beyond aerobic glycolysis: Transformed cells can engage in glutamine metabolism that exceeds the requirement for protein and nucleotide synthesis (Proceedings of the National Academy of Sciences of the United States of America)
  8. Real-time metabolic imaging. (Proceedings of the National Academy of Sciences of the United States of America)
  9. Glutamine and cancer: cell biology, physiology, and clinical opportunities. (Journal of Clinical Investigation)
  10. How cancer metabolism is tuned for proliferation and vulnerable to disruption (Nature)

Their papers