Vivian W. Chow
Physiology · 2010
A researcher in the Science corpus: 1 paper; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Physiology — 16 citations into it
Whom Vivian W. Chow trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Papers from Vivian W. Chow's perspective
Ordered by how strongly the researchers Vivian W. Chow trusts endorse each paper. Same corpus, a different order for every researcher.
- A disintegrin-metalloproteinase prevents amyloid plaque formation and hippocampal defects in an Alzheimer disease mouse model. (Journal of Clinical Investigation)
- Mice deficient in BACE1, the Alzheimer's β-secretase, have normal phenotype and abolished β-amyloid generation (Nature Neuroscience)
- Identification of the major Aβ1–42-degrading catabolic pathway in brain parenchyma: Suppression leads to biochemical and pathological deposition (Nature Medicine)
- Amyloid Precursor Protein Trafficking, Processing, and Function* (Journal of Biological Chemistry)
- Cellular Prion Protein Mediates Impairment of Synaptic Plasticity by Amyloid-β Oligomers (Nature)
- Naturally secreted oligomers of amyloid β protein potently inhibit hippocampal long-term potentiation in vivo (Nature)
- The disintegrin/metalloprotease ADAM 10 is essential for Notch signalling but not for alpha-secretase activity in fibroblasts.
- Alzheimer's disease: genes, proteins, and therapy. (Physiological Reviews)
- Common Structure of Soluble Amyloid Oligomers Implies Common Mechanism of Pathogenesis (Science)
- Formation of Neurofibrillary Tangles in P301L Tau Transgenic Mice Induced by Aβ42 Fibrils (Science)
Their papers
- An Overview of APP Processing Enzymes and Products (2010) — 645 citations