M. Gleichmann

Physiology · 2008–2010

A researcher in the Science corpus: 2 papers; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.

Subfields

Whom M. Gleichmann trusts

Their strongest outbound trust, strongest first, inferred from their citations.

  1. G. Thinakaran (in Physiology)

Papers from M. Gleichmann's perspective

Ordered by how strongly the researchers M. Gleichmann trusts endorse each paper. Same corpus, a different order for every researcher.

  1. A disintegrin-metalloproteinase prevents amyloid plaque formation and hippocampal defects in an Alzheimer disease mouse model. (Journal of Clinical Investigation)
  2. Mice deficient in BACE1, the Alzheimer's β-secretase, have normal phenotype and abolished β-amyloid generation (Nature Neuroscience)
  3. Identification of the major Aβ1–42-degrading catabolic pathway in brain parenchyma: Suppression leads to biochemical and pathological deposition (Nature Medicine)
  4. Amyloid Precursor Protein Trafficking, Processing, and Function* (Journal of Biological Chemistry)
  5. Cellular Prion Protein Mediates Impairment of Synaptic Plasticity by Amyloid-β Oligomers (Nature)
  6. Naturally secreted oligomers of amyloid β protein potently inhibit hippocampal long-term potentiation in vivo (Nature)
  7. Alzheimer's disease: genes, proteins, and therapy. (Physiological Reviews)
  8. The disintegrin/metalloprotease ADAM 10 is essential for Notch signalling but not for alpha-secretase activity in fibroblasts.
  9. Common Structure of Soluble Amyloid Oligomers Implies Common Mechanism of Pathogenesis (Science)
  10. Faculty Opinions recommendation of Two transmembrane aspartates in presenilin-1 required for presenilin endoproteolysis and gamma-secretase activity.

Their papers