J. Lotharius

Neurology · 2002

A researcher in the Science corpus: 1 paper; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.

Subfields

Whom J. Lotharius trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. M. Feany

Papers from J. Lotharius's perspective

Ordered by how strongly the researchers J. Lotharius trusts endorse each paper. Same corpus, a different order for every researcher.

  1. A Drosophila model of Parkinson's disease (Nature)
  2. Dopaminergic loss and inclusion body formation in alpha-synuclein mice: implications for neurodegenerative disorders. (Science)
  3. Chronic systemic pesticide exposure reproduces features of Parkinson's disease (Nature Neuroscience)
  4. Acceleration of oligomerization, not fibrillization, is a shared property of both alpha-synuclein mutations linked to early-onset Parkinson's disease: implications for pathogenesis and therapy. (Proceedings of the National Academy of Sciences of the United States of America)
  5. Proteasomal function is impaired in substantia nigra in Parkinson's disease. (Neuroscience Letters)
  6. Impairment of the ubiquitin-proteasome system by protein aggregation. (Science)
  7. Fibrils formed in vitro from alpha-synuclein and two mutant forms linked to Parkinson's disease are typical amyloid. (Biochemistry)
  8. Parkin functions as an E2-dependent ubiquitin- protein ligase and promotes the degradation of the synaptic vesicle-associated protein, CDCrel-1. (Proceedings of the National Academy of Sciences of the United States of America)
  9. Inherent toxicity of aggregates implies a common mechanism for protein misfolding diseases (Nature)
  10. In vivo positron emission tomographic evidence for compensatory changes in presynaptic dopaminergic nerve terminals in Parkinson's disease (Annals of Neurology)

Their papers