Pathogenesis of parkinson's disease: dopamine, vesicles and α-synuclein

By J. Lotharius, P. Brundin. Nature Reviews Neuroscience, 2002

906 citations recorded by the source corpus.

Subfields: Cellular and Molecular Neuroscience, Neurology

View Paper: https://www.semanticscholar.org/paper/9f0a2945e1b8c28932d6c8b1b03bcaf18fa09798

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cellular and Molecular Neuroscience

1. Jianzhi Zeng
2. A. Kreitzer
3. Wolfram Schultz
4. R. Malenka

In Neurology

1. A. Kreitzer
2. R. Malenka

Citations in the corpus

• Oxidative stress in the aging substantia nigra and the etiology of Parkinson's disease (2019, background): “…ciated with aberrant posttranslational modification or mutation of α‐synuclein protein, which can impede VMAT2‐mediated repack‐ aging of dopamine into synaptic vesicles (Lotharius & Brundin, 2002), and impair regulation of DAT expression at the cell surface (Sidhu, Wersinger, & Vernier, 2004).”
• A genetically-encoded fluorescent sensor enables rapid and specific detection of dopamine in flies, fish, and mice (2018, background): “, 1995), schizophrenia (Howes and Kapur, 2009), and Parkinson’s disease (Lotharius and Brundin, 2002).”