P. Mao
Physiology · 2011
A researcher in the Science corpus: 1 paper; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Physiology — 23 citations into it
Whom P. Mao trusts
Their strongest outbound trust, strongest first, inferred from their citations.
- D. Selkoe (in Physiology)
Papers from P. Mao's perspective
Ordered by how strongly the researchers P. Mao trusts endorse each paper. Same corpus, a different order for every researcher.
- Mitochondrial Abnormalities in Alzheimer's Disease (Journal of Neuroscience)
- Alzheimer's disease: genes, proteins, and therapy. (Physiological Reviews)
- Alzheimer's Disease Is a Synaptic Failure (Science)
- Nitric oxide‐induced mitochondrial fission is regulated by dynamin‐related GTPases in neurons (EMBO Journal)
- Cyclophilin D deficiency attenuates mitochondrial and neuronal perturbation and ameliorates learning and memory in Alzheimer's disease (Nature Medicine)
- Intracellular amyloid-beta in Alzheimer's disease. (Nature Reviews Neuroscience)
- Impaired mitochondrial dynamics and abnormal interaction of amyloid beta with mitochondrial protein Drp1 in neurons from patients with Alzheimer's disease: implications for neuronal damage. (Human Molecular Genetics)
- Mitochondria are a direct site of A beta accumulation in Alzheimer's disease neurons: implications for free radical generation and oxidative damage in disease progression. (Human Molecular Genetics)
- Inflammation in Alzheimer disease: driving force, bystander or beneficial response? (Nature Medicine)
- Amyloid-β overproduction causes abnormal mitochondrial dynamics via differential modulation of mitochondrial fission/fusion proteins (Proceedings of the National Academy of Sciences of the United States of America)