G. Krafft
Physiology · 2001–2004
A researcher in the Science corpus: 5 papers; trusts 1 researcher and is trusted by 5 researchers here, as inferred from the citation graph.
Subfields
- Physiology — 40 citations into it; #741 of 2,276 by trust here, #585 by citations
Whom G. Krafft trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts G. Krafft overall
Papers from G. Krafft's perspective
Ordered by how strongly the researchers G. Krafft trusts endorse each paper. Same corpus, a different order for every researcher.
- Targeting small Aβ oligomers: the solution to an Alzheimer's disease conundrum?
- Common Structure of Soluble Amyloid Oligomers Implies Common Mechanism of Pathogenesis (Science)
- Naturally secreted oligomers of amyloid β protein potently inhibit hippocampal long-term potentiation in vivo (Nature)
- Intraneuronal Aβ42 Accumulation in Human Brain
- Immunization reverses memory deficits without reducing brain Aβ burden in Alzheimer's disease model (Nature Neuroscience)
- Triple-transgenic model of Alzheimer's disease with plaques and tangles: intracellular Abeta and synaptic dysfunction. (Neuron)
- Pathways towards and away from Alzheimer's disease (Nature)
- Deciphering the molecular basis of memory failure in Alzheimer's disease. (Neuron)
- Morphological changes in dendritic spines associated with long-term synaptic plasticity. (Annual Review of Neuroscience)
- Inhibition of Activity-Dependent Arc Protein Expression in the Rat Hippocampus Impairs the Maintenance of Long-Term Potentiation and the Consolidation of Long-Term Memory (Journal of Neuroscience)
Their papers
- Alzheimer's disease-affected brain: Presence of oligomeric Aβ ligands (ADDLs) suggests a molecular basis for reversible memory loss (2003) — 1,118 citations
- Synaptic Targeting by Alzheimer's-Related Amyloid β Oligomers (2004) — 861 citations
- In Vitro Characterization of Conditions for Amyloid-β Peptide Oligomerization and Fibrillogenesis* (2003) — 1,002 citations
- Oligomeric and Fibrillar Species of Amyloid-β Peptides Differentially Affect Neuronal Viability* (2002) — 1,466 citations
- Targeting small Aβ oligomers: the solution to an Alzheimer's disease conundrum? (2001) — 1,021 citations