James L. Riley

Immunology · 2009

A researcher in the Science corpus: 1 paper; trusts 12 researchers and is trusted by 5 researchers here, as inferred from the citation graph.

Subfields

Whom James L. Riley trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Taku Okazaki
  2. Arlene H. Sharpe
  3. G. Freeman
  4. R. Parry
  5. B. Fife
  6. R. Abraham
  7. E. Wherry
  8. M. Butte
  9. Tasuku Honjo
  10. Kenneth A. Frauwirth

Who trusts James L. Riley overall

  1. D. Chaplin
  2. Loise M. Francisco
  3. Arlene H. Sharpe
  4. Jaikumar Duraiswamy
  5. Kristen E. Pauken
  6. Michael Loran Dustin
  7. Joel Crespo

Papers from James L. Riley's perspective

Ordered by how strongly the researchers James L. Riley trusts endorse each paper. Same corpus, a different order for every researcher.

  1. PD-1 immunoreceptor inhibits B cell receptor-mediated signaling by recruiting src homology 2-domain-containing tyrosine phosphatase 2 to phosphotyrosine (Proceedings of the National Academy of Sciences of the United States of America)
  2. SHP-1 and SHP-2 Associate with Immunoreceptor Tyrosine-Based Switch Motif of Programmed Death 1 upon Primary Human T Cell Stimulation, but Only Receptor Ligation Prevents T Cell Activation1 (Journal of Immunology)
  3. The PD-1-PD-L pathway in immunological tolerance. (Trends in immunology)
  4. Programmed death-1 ligand 1 interacts specifically with the B7-1 costimulatory molecule to inhibit T cell responses. (Immunity)
  5. PD‐1:PD‐L inhibitory pathway affects both CD4+ and CD8+ T cells and is overcome by IL‐2 (European Journal of Immunology)
  6. The B7 family revisited. (Annual Review of Immunology)
  7. Restoring function in exhausted CD8 T cells during chronic viral infection (Nature)
  8. The CD28 signaling pathway regulates glucose metabolism. (Immunity)
  9. PD-L2 is a second ligand for PD-1 and inhibits T cell activation (Nature Immunology)
  10. CTLA-4 and PD-1 Receptors Inhibit T-Cell Activation by Distinct Mechanisms (Molecular and Cellular Biology)

Their papers