Ulrich Hämmerling
Cell Metabolism · 2005
A researcher in the Science corpus: 1 paper; trusts 0 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Papers from Ulrich Hämmerling's perspective
Ordered by how strongly the researchers Ulrich Hämmerling trusts endorse each paper. Same corpus, a different order for every researcher.
- Complex III Releases Superoxide to Both Sides of the Inner Mitochondrial Membrane* (Journal of Biological Chemistry)
- Faculty Opinions recommendation of Targeting of HIF-alpha to the von Hippel-Lindau ubiquitylation complex by O2-regulated prolyl hydroxylation.
- Redistribution of Intracellular Oxygen in Hypoxia by Nitric Oxide: Effect on HIF1α (Science)
- Reactive Oxygen Species Generated at Mitochondrial Complex III Stabilize Hypoxia-inducible Factor-1α during Hypoxia (Journal of Biological Chemistry)
- Stable suppression of gene expression by RNAi in mammalian cells (Proceedings of the National Academy of Sciences of the United States of America)
- Mitochondrial dysfunction resulting from loss of cytochrome c impairs cellular oxygen sensing and hypoxic HIF-alpha activation. (Cell Metabolism)
- Characterization of the Human Prolyl 4-Hydroxylases That Modify the Hypoxia-inducible Factor* (Journal of Biological Chemistry)
- Oxygen sensing requires mitochondrial ROS but not oxidative phosphorylation. (Cell Metabolism)
- HIF prolyl‐hydroxylase 2 is the key oxygen sensor setting low steady‐state levels of HIF‐1α in normoxia (EMBO Journal)
- C. elegans EGL-9 and mammalian homologs define a family of dioxygenases that regulate HIF by prolyl hydroxylation. (Cell)