K. Wolter
Physiology · 2015–2021
A researcher in the Science corpus: 3 papers; trusts 2 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Physiology — 42 citations into it
Whom K. Wolter trusts
Their strongest outbound trust, strongest first, inferred from their citations.
- J. Campisi (in Physiology)
- Jan M. van Deursen (in Physiology)
Papers from K. Wolter's perspective
Ordered by how strongly the researchers K. Wolter trusts endorse each paper. Same corpus, a different order for every researcher.
- Senescence and tumour clearance is triggered by p53 restoration in murine liver carcinomas (Nature)
- Oncogene-induced senescence is a DNA damage response triggered by DNA hyper-replication (Nature)
- Chemokine signaling via the CXCR2 receptor reinforces senescence. (Cell)
- Senescence of activated stellate cells limits liver fibrosis (Cell)
- Lamin B1 loss is a senescence-associated biomarker (Molecular Biology of the Cell)
- Senescence-messaging secretome: SMS-ing cellular stress (Nature Reviews. Cancer)
- Clearance of p16Ink4a-positive senescent cells delays ageing-associated disorders (Nature)
- p38MAPK is a novel DNA damage response‐independent regulator of the senescence‐associated secretory phenotype (EMBO Journal)
- Inflammaging and anti-inflammaging: a systemic perspective on aging and longevity emerged from studies in humans. (Mechanisms of Ageing and Development)
- Rapamycin fed late in life extends lifespan in genetically heterogeneous mice (Nature)
Their papers
- mTOR regulates MAPKAPK2 translation to control the senescence-associated secretory phenotype (2015) — 708 citations
- NASH limits anti-tumour surveillance in immunotherapy-treated HCC (2021) — 1,066 citations
- Innate immune sensing of cytosolic chromatin fragments through cGAS promotes senescence (2017) — 1,037 citations