W. Cui
Molecular Biology · with Yu · 2016
A researcher in the Science corpus: 1 paper; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 38 citations into it
Whom W. Cui trusts
Their strongest outbound trust, strongest first, inferred from their citations.
- David M. Sabatini (in Molecular Biology)
Papers from W. Cui's perspective
Ordered by how strongly the researchers W. Cui trusts endorse each paper. Same corpus, a different order for every researcher.
- S6K1−/−/S6K2−/− Mice Exhibit Perinatal Lethality and Rapamycin-Sensitive 5′-Terminal Oligopyrimidine mRNA Translation and Reveal a Mitogen-Activated Protein Kinase-Dependent S6 Kinase Pathway (Molecular and Cellular Biology)
- Prolonged rapamycin treatment inhibits mTORC2 assembly and Akt/PKB. (Molecules and Cells)
- Induction of pluripotent stem cells from mouse embryonic and adult fibroblast cultures by defined factors.
- Insulin Resistance and a Diabetes Mellitus-Like Syndrome in Mice Lacking the Protein Kinase Akt2 (PKBβ)
- SIN1/MIP1 maintains rictor-mTOR complex integrity and regulates Akt phosphorylation and substrate specificity. (Cell)
- mTOR signaling in growth control and disease (Cell)
- The ground state of embryonic stem cell self-renewal (Nature)
- mTOR Is Essential for Growth and Proliferation in Early Mouse Embryos and Embryonic Stem Cells (Molecular and Cellular Biology)
- Promotion of Reprogramming to Ground State Pluripotency by Signal Inhibition (PLoS Biology)
- Ablation in mice of the mTORC components raptor, rictor, or mLST8 reveals that mTORC2 is required for signaling to Akt-FOXO and PKCalpha, but not S6K1. (Developmental Cell)