Urmi Sengupta
Physiology · 2011–2016
A researcher in the Science corpus: 2 papers; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Physiology — 31 citations into it
- Molecular Biology — 18 citations into it
- Neurology — 10 citations into it
Whom Urmi Sengupta trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Papers from Urmi Sengupta's perspective
Ordered by how strongly the researchers Urmi Sengupta trusts endorse each paper. Same corpus, a different order for every researcher.
- Neuropathological stageing of Alzheimer-related changes
- S3‐03‐05: Transmission and Spreading of Tauopathy in Transgenic Mouse Brain
- Staging of Alzheimer disease-associated neurofibrillary pathology using paraffin sections and immunocytochemistry (Acta Neuropathologica)
- Propagation of Tau Misfolding from the Outside to the Inside of a Cell* (Journal of Biological Chemistry)
- Cellular Prion Protein Mediates Impairment of Synaptic Plasticity by Amyloid-β Oligomers (Nature)
- A mutation in APP protects against Alzheimer’s disease and age-related cognitive decline (Nature)
- Protein misfolding, functional amyloid, and human disease. (Annual Review of Biochemistry)
- Tau-mediated neurodegeneration in Alzheimer's disease and related disorders (Nature Reviews Neuroscience)
- Toward defining the preclinical stages of Alzheimer's disease: Recommendations from the National Institute on Aging-Alzheimer's Association workgroups on diagnostic guidelines for Alzheimer's disease (Alzheimer's & Dementia)
- Proliferation of amyloid-β42 aggregates occurs through a secondary nucleation mechanism (Proceedings of the National Academy of Sciences of the United States of America)
Their papers
- The Role of Amyloid-β Oligomers in Toxicity, Propagation, and Immunotherapy (2016) — 668 citations
- Tau oligomers impair memory and induce synaptic and mitochondrial dysfunction in wild-type mice (2011) — 597 citations