ERK and cell death: Mechanisms of ERK‐induced cell death – apoptosis, autophagy and senescence
By S. Cagnol, J. Chambard. The FEBS Journal, 2010
1,271 citations recorded by the source corpus.
Subfields: Epidemiology, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/fd99b62e17565878549c010d19759af1d48b90f4
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Citations in the corpus
- Anti-apoptosis and cell survival: a review. (2011, background): “294], a number of different types of receptors including GPCRs, steroids, cytokines and receptor tyrosine kinases [65,71,200,295– 300], enzymes including a number of kinases such as ERK1/2 as well as a varieties others such as sphingomyelin synthase and dUTPase [100,101,113,156,280,301], integrins a”
- Understanding MAPK Signaling Pathways in Apoptosis (2020, background): “It has been proposed that the main cause of sustained ERK activation is the presence of ROS during the cell death program [68].”
- Signal transduction in cancer. (2015, neutral): “4) (review Cagnol and Chambard 2010; Zhang et al. 2011).”