Regulation of muscle mass by myostatin.

By Se-Jin Lee. Annual Review of Cell and Developmental Biology, 2004

839 citations recorded by the source corpus.

Subfields: Cardiology and Cardiovascular Medicine, Molecular Biology, Surgery

View Paper: https://www.semanticscholar.org/paper/fd4f2b685f3a8e118e01add20ad1b4127269de97

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cardiology and Cardiovascular Medicine

1. S. Schiaffino
2. P. Bonaldo
3. Duojia Pan
4. M. Sandri

In Molecular Biology

1. M. Sandri
2. S. Schiaffino
3. P. Bonaldo
4. B. Blaauw
5. Duojia Pan
6. K. Guan
7. I. Slaninová
8. Bin Zhao

In Surgery

1. P. Bonaldo
2. Duojia Pan
3. M. Sandri

Citations in the corpus

• MicroRNA-208a is a regulator of cardiac hypertrophy and conduction in mice. (2009, background): “Myostatin stood out as an interesting candidate, given its role as a repressor of hypertrophic growth in skeletal muscle (36, 37).”
• Angiotensin II type 1 receptor blockade attenuates TGF-β–induced failure of muscle regeneration in multiple myopathic states (2007, background): “One complicating factor in interpretation of these data is that myostatin (encoded by Mstn ), another member of the TGF-β superfamily that suppresses satellite cell activit”
• Mechanisms regulating skeletal muscle growth and atrophy (2013, background): “79 Lee SW, Dai G, Hu Z, Wang X, Du J & Mitch WE (2004) Regulation of muscle protein degradation: coordinated control of apoptotic and ubiquitin– proteasome systems by phosphatidylinositol 3 kinase.”
• Hippo signaling in organ size control. (2007, background): “A noted precedent for such paracrine signaling in organ size control can be found in myostatin, a TGF- family protein that is secreted by muscle cells to inhibit their own growth (Lee 2004).”
• Cellular and molecular mechanisms of muscle atrophy (2013, background): “The increase in muscle mass caused by loss of myostatin is a consequence of both hyperplasia (i.e. increased cell number) and hypertrophy (i.e. increased cell size) (Lee, 2004; Lee and McPherron, 1999; McPherron et al., 1997).”
• An official American Thoracic Society/European Respiratory Society statement: update on limb muscle dysfunction in chronic obstructive pulmonary disease. (2014, background): “transforming growth factor-b (TGF-b) family member that acts as a negative regulator of limb muscle growth (171, 172),”