Mind blindness and the brain in autism.

By Uta Frith. Neuron, 2001

618 citations recorded by the source corpus.

Subfields: Cognitive Neuroscience, Developmental and Educational Psychology

View Paper: https://www.semanticscholar.org/paper/fc2632a1136518d7107ca2b47097b74aebce691a

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Citations in the corpus

The developing brain and neural plasticity: Implications for normality, psychopathology, and resilience. (2015, background): “In addition, Frith (2001) has proposed a somewhat controversial etiological theory of Autism linking deficits in theory of mind (“mind blindness”) to abnormalities in a network of brain regions, including the medial prefrontal cortex, areas in the temporal-parietal region, and the temporal poles,…”
The neural basis of human moral cognition (2005, background): “The posterior STS is a key region for storing these representation”
Understanding Others: Brain Mechanisms of Theory of Mind and Empathy (2008, background): “The lack of a ToM in most autistic children could explain their observed failure in communication and social interaction (for a review, see Frith, 2001).”
The Enigmatic temporal pole: a review of findings on social and emotional processing. (2007, background): “These findings have led some researchers to conclude that the temporal poles are a critical part of the ‘mentalizing’ brain (Frith, 2001; Frith and Frith, 2003).”
Development of the adolescent brain: implications for executive function and social cognition. (2006, background): “In addition to executive functions, there is evidence that the prefrontal cortex is involved in several other high-level cognitive capacities, including selfawareness (Ochsner, 2004) and theory of mind (Frith & Frith, 2003), that is the ability to understand other minds by attributing mental states ”
The Cognitive Neuroscience of Moral Judgment (2010, background): “The aforementioned regions are implicated in emotional processing (Maddock, 1999; Phan et al., 2002; Adolphs, 2003) as well as in “theory of mind” (ToM) (Frith, 2001; Adolphs, 2003; Amodio and Frith, 2006; Young et al., 2007).”
Autism Spectrum Disorder and Specific Language Impairment: Overlaps in Syntactic Profiles (2016, background): “Given this, various researchers have suggested that communicative and social difficulties associated with this population are the result of their difficulties with mentalizing and perspective switching (Frith, Morton & Leslie 1991; Frith 2001; Tager-Flusberg 2007).”
Understanding autism: insights from mind and brain. (2003, background): “In this way relationships between specific brain function and behaviour have been investigated (for a review, see Frith (2001)).”
Evaluating the theory of executive dysfunction in autism (2004, background): “Aspects of the other two main cognitive theories of autism have been found in the broader phenotype providing good evidence for its existence across broad areas of its features (e.g. Baron-Cohen & Hammer, 1997; Happé, Briskman, & Frith, 2001).”
Activations related to "mirror" and "canonical" neurones in the human brain: an fMRI study (2003, background): “…as the superior temporal sulcus receives input from both dorsal and ventral streams (Baizer et al., 1991), it could have a role in the extraction of contextual and intentional cues (Toni et al., 2001; Frith, 2001) which are necessary for various tasks, including visuomotor transformations.”
Social and emotional attachment in the neural representation of faces (2004, neutral): “…imaging studies have found consistently that the anterior paracingulate cortex and the posterior superior temporal sulcus are activated during the performance of tasks that require interpreting and predicting other people's behavior (Castelli et al., 2000; Frith, 2001; Gallagher et al., 2000).”
The rises and falls of disconnection syndromes. (2005, background): “…disconnection paradigm is still to be found within the neurology clinic and outside it within ‘functional’ disorders as diverse as schizophrenia (Bullmore et al., 1997), autism (Frith, 2001) and dyslexia (Demonet et al., 2004), where disconnecting ‘lesions’ remain inferred rather than demonstrable.”