Vitamin D and human health: lessons from vitamin D receptor null mice.
By R. Bouillon, G. Carmeliet, L. Verlinden, E. van Etten, A. Verstuyf, Hilary F Luderer, L. Lieben, C. Mathieu, M. Demay. Endocrine reviews, 2008
1,789 citations recorded by the source corpus.
Subfields: Genetics, Health, Toxicology and Mutagenesis, Pathology and Forensic Medicine
View Paper: https://www.semanticscholar.org/paper/fbb87f33ae4c313880fb27642f2003fd23545b3d
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Citations in the corpus
- The nonskeletal effects of vitamin D: an Endocrine Society scientific statement. (2012, background): “For example, VDR null mice exhibit atrophy of adipose tissue in mammary and prostate glands (94, 102).”
- Vitamin D: The “sunshine” vitamin (2012, background): “[64] Studies have, however, shown that many other cell types, including those of the vascular wall, express 1α-hydroxylase with subsequent intracellular conversion of 25(OH)D to 1,25(OH)2D, which exerts its effects at the level of the individual cell or tissue before being catabolized to biologicall”
- Vitamin D and cardiovascular disease. (2014, background): “Structurally, VDR consists of a short N-terminal domain before the DNA-binding domain comprising 2 zinc fingers, the first conferring vitamin D response element specificity and the second providing a site for heterodimerization.2 The rest of the molecule contains the lipophilic 1,25(OH) 2 D- binding”
- Vitamin D and Immune Function (2013, background): “First evidence for the positive effect of vitamin D intake for human health came from early studies on rickets and osteomalacia (reviewed in [1]).”