Graph theoretical analysis of magnetoencephalographic functional connectivity in Alzheimer's disease.

By C. Stam, W. D. Haan, A. Daffertshofer, B. Jones, Ilonka Manshanden, A. V. C. Walsum, T. Montez, Jeroen P. A. Verbunt, J. Munck, B. W. Dijk and 2 more. Brain : a journal of neurology, 2009

886 citations recorded by the source corpus.

Subfields: Cognitive Neuroscience

View Paper: https://www.semanticscholar.org/paper/f96f659ada20f35e9fce66306c8f8f6a471e592d

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In Cognitive Neuroscience

Citations in the corpus

Alpha-synuclein in Parkinson’s disease and other synucleinopathies: from overt neurodegeneration back to early synaptic dysfunction (2023, background): “Neuropathological evidence from PD patients shows that dopaminergic terminals in the putamen are lost significantly earlier than neuron soma [86].”
Role of neuroinflammation in neurodegeneration development (2023, background): “important for motor functions, so PD is clinically characterized by a range of motor symptoms.(50) The onset of disease usually occurs decades before the first symptom appears.”
The Search for True Numbers of Neurons and Glial Cells in the Human Brain: A Review of 150 Years of Cell Counting (2016, background): “The Journal of Comparative Neurology | Research in Systems Neuroscience 3881 classical studies documenting the loss of neurons in degenerative diseases, for example correlating the extent of neuron loss with disease severity (Damier et al., 1999; Stark and Pakkenberg, 2004; Kordower et al., 2013).”
The Parkinson's progression markers initiative (PPMI) – establishing a PD biomarker cohort (2018, background): “First, there is increasing evidence that ongoing progression may lessen any potential therapeutic effect, as pathologic studies indicate significant dopaminergic degeneration present already at 4 years postdiagnosis.(35) Second, studies designed to evaluate diseasemodifying therapeutics are limited ”
Functions of plasmalogen lipids in health and disease. (2012, background): “It may be both, as demonstrated by the finding that plasmalogen deficiency further aggravates brain injury in the X-ALD mouse model [81].”
Focal Brain Lesions to Critical Locations Cause Widespread Disruption of the Modular Organization of the Brain (2012, neutral): “As described above, regions with less than 25% EPI coverage within an ROI in either hemisphere in any single participant in a group were excluded, resulting in 87 regions in controls and 86 regions in lesion patients in each hemisphere.”