Molecular mechanisms of glioma invasiveness: the role of proteases
By J. S. Rao. Nature Reviews. Cancer, 2003
877 citations recorded by the source corpus.
Subfields: Cancer Research, Molecular Biology, Oncology
View Paper: https://www.semanticscholar.org/paper/f790e9b5ab75fdad30c660c443ac7d48b38f340c
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In Cancer Research
- J. Joyce
- D. Hanahan
- Zena Werb
- G. Bergers
- Guido Kroemer
- Lisa M. Coussens
- Nicole Meyer-Morse
- Richard J. Johnson
In Molecular Biology
- D. Hanahan
- J. Joyce
- Zena Werb
- G. Bergers
- Guido Kroemer
- M. Jäättelä
- Nicole Meyer-Morse
- Richard J. Johnson
In Oncology
- D. Hanahan
- Zena Werb
- J. Joyce
- Guido Kroemer
- Nicole Meyer-Morse
- G. Bergers
- Bonnie F. Sloane
- M. Jäättelä
Citations in the corpus
- HIF1alpha induces the recruitment of bone marrow-derived vascular modulatory cells to regulate tumor angiogenesis and invasion. (2008, background): “Congruent with the observation that a variety of tumors express VEGFR1 or -2 and their neuropilin coreceptors (Lesslie et al., 2006; Rao, 2003 ), we found that GBM cells express VEGFR1 and -2 and neuropilin 1 and 2 and that addition of VEGF to tumor cells or the generation of tumor cells that ectop”
- Lysosomes and autophagy in cell death control (2005, background): “Tumour invasion and metastasis are associated with altered lysosomal trafficking and increased expression of cathepsins, especially the cysteine cathepsins — cathepsin B and cathepsin L — as well as the aspartate cathepsin, cathepsin”
- Cysteine cathepsins: multifunctional enzymes in cancer (2006, background): “The finding that five transcript variants have been identified for cathepsin B in human gliomas, a tumour type in which cathepsin B is highly expressed and in which downregulation of cathepsin B has been shown to reduce growth, invasion and angiogenesis (for a review, see Ref”
- Diffuse glioma growth: a guerilla war (2007, background): “excellent recent reviews on (some of) these aspects we refer the reader to [40, 57, 69, 70, 99, 137, 148, 172].”
- Cathepsin cysteine proteases are effectors of invasive growth and angiogenesis during multistage tumorigenesis. (2004, background): “Although their function is broadly antitumorigenic (Kond- ATGAGG), cathepsin S (forward: 5 - CGCCAGCCATTCCTCCTT-3 , reverse: uri et al., 2002; Rao, 2003), there have been recent reports 5 -ATGATTCACATTGCCCGTACAG-3 , probe: ACAAAAGCGGTGTCTATG ACGACCCC), cathepsin Z (forward: 5 -CCTGTCCGGGAGGGAGAA-3…”