The effects of physical activity on serum C-reactive protein and inflammatory markers: a systematic review.

By Christos Kasapis, P. Thompson. Journal of the American College of Cardiology, 2005

1,227 citations recorded by the source corpus.

Subfields: Cardiology and Cardiovascular Medicine, Epidemiology, Rehabilitation

View Paper: https://www.semanticscholar.org/paper/f6b7759868b2aaf5871ed0f902f46635899d1abc

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cardiology and Cardiovascular Medicine

1. B. K. Pedersen
2. R. Shephard

In Epidemiology

1. B. K. Pedersen
2. Haiming Cao

In Rehabilitation

1. B. K. Pedersen
2. R. Shephard
3. M. C. Severinsen
4. B. Saltin
5. C. Fischer
6. Haiming Cao

Citations in the corpus

• Rethinking IL-6 and CRP: Why they are more than inflammatory biomarkers, and why it matters. (2018, neutral): “The same delayed time course has been observed after surgery, physical trauma, and intense physical activity (e.g., Di Napoli et al., 2012; Ispirlidis et al., 2008; Kasapis & Thompson, 2005; Nguyen-Vermillion et al., 2011).”
• Anti-inflammatory effect of exercise training in subjects with type 2 diabetes and the metabolic syndrome is dependent on exercise modalities and independent of weight loss. (2010, background): “The dependence of hs-CRP reduction on intensity and type of exercise and duration of training program might explain the discordant results of previous studies in the general population [15,16] and in subjects with type 2 diabetes [19,20] with exercise protocols of different modalities and duration.”
• Position statement. Part one: Immune function and exercise. (2011, background): “Human cross-sectional studies demonstrate an inverse relationship between regular physical activity and inflammatory biomarkers, including C-reactive protein (CRP), tumour necrosis factor-alpha (TNF-α), and interleukin-6 (IL-6) (123, 225).”
• Obesity and C‐reactive protein in various populations: a systematic review and meta‐analysis (2013, background): “Multiple mechanisms have been suggested, including modification of cytokine production from skeletal muscles, endothelial and blood mononuclear cells, improved endothelial function and insulin sensitivity, and an antioxidant effect (37).”
• Exercise standards for testing and training: a scientific statement from the American Heart Association. (2013, background): “Many, but not all, cross-sectional epidemiological studies have reported an independent inverse association between physical activity level and hs-CRP levels over a broad range of physical activity levels, including ultra-marathon running.(452,453) The relationship might differ by type of physical a”
• Obesity and inflammation: the linking mechanism and the complications (2016, background): “Recent studies have shown a positive independent association between atherosclerotic events and CRP [52, 56, 57], which acts as a proatherosclerotic factor by up-regulating angiotensin type-1 receptor expression [58].”
• Metabolic Syndrome and Insulin Resistance: Underlying Causes and Modification by Exercise Training (2013, neutral): “Chronic exercise exerts potent anti-inflammatory effects (40, 335, 502) and these effects are likely mediated by direct effects on the immune system and a reduction in visceral fat including diminished release of proinflammatory cytokines and chemokines from adipocytes [as reviewed by reference (223”