The spectrum of disease in chronic traumatic encephalopathy.

By Ann C. McKee, R. Stern, Christopher J. Nowinski, T. Stein, Victor E. Alvarez, D. Daneshvar, Hyo-Soon Lee, Sydney M Wojtowicz, G. Hall, Christine M. Baugh and 13 more. Brain : a journal of neurology, 2013

2,007 citations recorded by the source corpus.

Subfields: Epidemiology, Neurology, Physiology

View Paper: https://www.semanticscholar.org/paper/23892cae5f831fad7065ad1613687c672fca499d

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Epidemiology

In Neurology

In Physiology

Citations in the corpus

Aging-related tau astrogliopathy (ARTAG): harmonized evaluation strategy (2015, neutral): “On the other hand, tau pathology, including neuronal and astroglial, in CTE is more abundant in the depths of the convexity cerebral sulci, especially in early stages [41], an aspect that has not been reported in tau astrogliopathy in the aging brain [29, 34-36, 46, 57].”
Clinicopathological Evaluation of Chronic Traumatic Encephalopathy in Players of American Football (2017, neutral): “Motor neuron disease 4 (9) 7 (5) 11 (6)”
Traumatic brain injuries (2016, methodology): “Patients with non-sports-related mild TBI more often have pre-existing behavioural, psychiatric and/or substance abuse problems that increase their susceptibility to injury and might prolong recovery7,8, although sports-related TBIs might result from lower mechanical forces than non-sports-related T”
The first NINDS/NIBIB consensus meeting to define neuropathological criteria for the diagnosis of chronic traumatic encephalopathy (2015, methodology): “Although 9 of the 10 subjects diagnosed with CTE in this study were former American football players and only one was a former professional boxer, previous data has shown that the pathological features of CTE associated with boxing (often referred to as “dementia pugilistica”) are similar to the pat”
Alzheimer’s and Parkinson’s diseases: The prion concept in relation to assembled Aβ, tau, and α-synuclein (2015, positive): “They are later found throughout cerebral cortex, diencephalon, basal ganglia, brainstem, and spinal cord (78).”
Pathology of Neurodegenerative Diseases. (2017, background): “…2003), dementia with Lewy bodies (DLB) (Kosaka et al. 1984), amyotrophic lateral sclerosis (ALS) (Brettschneider et al. 2013), frontotemporal lobar degeneration associated with TDP-43 pathology (FTLD-TDP) (Brettschneider et al. 2014), and chronic traumatic encephalopathy (CTE) (McKee et al. 2013).”