Insights into the evolution of Yersinia pestis through whole-genome comparison with Yersinia pseudotuberculosis.

By P. Chain, É. Carniel, F. Larimer, J. Lamerdin, P. Stoutland, W. Regala, A. Georgescu, Lisa M. Vergez, M. Land, V. Motin and 13 more. Proceedings of the National Academy of Sciences of the United States of America, 2004

657 citations recorded by the source corpus.

Subfields: Genetics, Molecular Biology, Parasitology

View Paper: https://www.semanticscholar.org/paper/f1df46ed4c1b19db7d79270142a12da7b7ba25c2

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Douglas R. Green

Citations in the corpus

Caspase-8 induces cleavage of gasdermin D to elicit pyroptosis during Yersinia infection (2018, background): “The more dangerous Y. pestis evolved from Y. pseudotuberculosis and in the process acquired a hypoacylated TLR4-evasive LPS (52).”
PathogenFinder - Distinguishing Friend from Foe Using Bacterial Whole Genome Sequence Data (2013, background): “Among the toxins found were: exofiliative toxin A (family-rank 5) in Staphylococcus aureus strains, causative of Staphylococcal scalded skin syndrome [43,44]; streptolysin (O and S), mainly found in Streptococcus pathogenic species [45]; hemolysin (II, III, a and b types) found in PFs mainly compose”
Autoinducer 2 Controls Biofilm Formation in Escherichia coli through a Novel Motility Quorum-Sensing Regulator (MqsR, B3022) (2006, background): “MqsR appears to be a conserved regulator protein (98 amino acids), since it has 50% homology with hypothetical proteins from Yersinia pseudotuberculosis, Yersinia pestis, Cupriavidus oxalaticus, Bordetella bronchiseptica, Pseudomonas fluorescens, and Bordetella pertussis (8, 27, 29, 43, 48).”