Innate partnership of HLA-B and KIR3DL1 subtypes against HIV-1

By Maureen P. Martin, Y. Qi, Xiaojiang Gao, Eriko Yamada, Jeffrey N. Martin, F. Pereyra, S. Colombo, E. Brown, W. Shupert, J. Phair and 11 more. Nature Genetics, 2007

778 citations recorded by the source corpus.

Subfields: Immunology

View Paper: https://www.semanticscholar.org/paper/f11c5851aaea80982e116a5dfe11412e1130e907

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In Immunology

1. Astrid Krmpotić
2. S. Jonjić
3. V. J. Lisnić
4. A. McMichael

Citations in the corpus

• Modulation of natural killer cell activity by viruses (2010, positive): “Other examples of virus-driven diversification of receptors include differential expression of an activating KIR3DS1 which leads to a better control of HIV virus [37], higher IFN-g and CD107a expression in unstimulated NK cells in individuals possessing KIR3DS1 gene [38], influence of distinct allel”
• Major Histocompatibility Complex Genomics and Human Disease (2013, neutral): “Combinations of HLA and KIR alleles have been associated with viral infection, malaria, autoimmune disease, cancer, transplantation, and complications of pregnancy (46, 48, 77, 79, 124).”
• The immune response during acute HIV-1 infection: clues for vaccine development (2009, background): “, which are thought to be ligands for these receptor”
• A random forest approach to the detection of epistatic interactions in case-control studies (2009, background): “Biomedical studies have also been confirming the existence of epistasis in many complex diseases, including myocardial infarction [4], breast cancer [5], hypertension [6], atrial fibrillation [7], diabetes mellitus type 2 [8], AIDS [9], and many others.”