Update of Neuropathology and Neurological Recovery After Traumatic Brain Injury

By J. Povlishock, D. Katz. The journal of head trauma rehabilitation, 2005

705 citations recorded by the source corpus.

Subfields: Epidemiology, Molecular Biology, Neurology

View Paper: https://www.semanticscholar.org/paper/f0d339612574bf0afe6ae43ff4d4bcd3d8b77dde

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Epidemiology

1. Henrik Zetterberg
2. Ann C. McKee

In Molecular Biology

1. Henrik Zetterberg
2. Kaj Blennow
3. Ann C. McKee

In Neurology

1. Henrik Zetterberg
2. Kaj Blennow
3. Ann C. McKee

Citations in the corpus

• White matter damage and cognitive impairment after traumatic brain injury (2010, background): “The pathology of diffuse axonal injury has been investigated in some detail (Povlishock and Katz, 2005), but, until recently, it has been difficult to study the location and extent of this damage, or its functional consequences in vivo.”
• Traumatic brain injuries (2016, background): “The mechanisms underlying this neurofilament compaction include disruption of the integrity of the plasma membrane leading to increased intracellular calcium, activation of calpain and caspase, and cytoskeletal proteolysis including spectrin and neurofilament side arm cleavage.(58) Another form of a”
• Role of Microglia in Neurotrauma (2010, neutral): “Mechanical forces at the moment of traumatic brain injury (TBI) cause rapid tissue deformation, resulting in primary physical damage.(116) The mechanisms involved in cell death and tissue loss following TBI are complex interactions between acute and delayed biochemical, molecular, and physiological ”
• Axonal Pathology in Traumatic Brain Injury (2012, background): “…(DAI) (Adams, et al., 1989, Adams, et al., 1982, Graham, et al., 1988, Povlishock, 1992, Povlishock and Becker, 1985, Povlishock, et al., 1983, Povlishock and Katz, 2005, Smith and Meaney, 2000), which may reflect the selective vulnerability of white matter axons to damage from mechanical…”
• Classification of Traumatic Brain Injury for Targeted Therapies (2008, neutral): “Numerous postmortem studies and imaging studies have shown that there are four main pathoanatomical sequelae of TBI: contusions; SAH; hematomas, including epidural, subdural, and intraparenchymal lesions; and DAI (Fig.”