Comparison of community- and health care-associated methicillin-resistant Staphylococcus aureus infection.

By T. Naimi, K. Ledell, K. Como‐Sabetti, S. Borchardt, D. Boxrud, J. Etienne, Susan K. Johnson, F. Vandenesch, S. Fridkin, C. O'Boyle and 2 more. Journal of the American Medical Association (JAMA), 2003

1,842 citations recorded by the source corpus.

Subfields: Infectious Diseases, Molecular Biology, Public Health, Environmental and Occupational Health

View Paper: https://www.semanticscholar.org/paper/ef9cab6b6b22668de56446491971a39aaf40a09b

How researchers see this paper

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In Infectious Diseases

1. Victor J. Torres
2. Francis Alonzo
3. Kunyan Zhang
4. F. Tenover
5. E. Stobberingh
6. R. H. Deurenberg
7. D. Levine
8. Robert S. Daum

In Molecular Biology

1. Victor J. Torres
2. Francis Alonzo
3. E. Stobberingh
4. R. H. Deurenberg
5. Henry F. Chambers
6. Robert S. Daum
7. Michael Z. David
8. Kunyan Zhang

In Public Health, Environmental and Occupational Health

1. Victor J. Torres
2. S. Cosgrove
3. Henry F. Chambers
4. Robert S. Daum
5. Michael Z. David
6. Francis Alonzo
7. Kunyan Zhang
8. Sahreena S. Lakhundi

Citations in the corpus

• The molecular evolution of methicillin-resistant Staphylococcus aureus. (2007, background): “Although at least eight definitions for CAMRSA exist [94,95], a general and international agreement has now been reached on a universal definition of CA-MRSA, i.”
• The Bicomponent Pore-Forming Leucocidins of Staphylococcus aureus (2014, background): “In fact, the predominant strain causing community-acquired disease in the United States, USA300, contains the prophage-carried PVL genes in the majority of isolates (128, 130).”
• Clinical practice guidelines by the infectious diseases society of america for the treatment of methicillin-resistant Staphylococcus aureus infections in adults and children: executive summary. (2011, background): “However, because 95%–100% of CA-MRSA strains are susceptible in vitro [81, 82], it has become an important option for the outpatient treatment of SSTI [83–85].”
• Staphylococcus aureus endocarditis: a consequence of medical progress. (2005, result): “Community-acquired MRSA cases differ demographically from health care–associated MRSA cases; MRSA isolates from community-acquired infections commonly possess distinct exotoxin gene profiles (eg, PantonValentine leukocidin genes) compared with health care–associated isolates.(20) The findings of the”
• The evolution of Staphylococcus aureus. (2008, neutral): “CA-MRSA is defined in several ways (Naimi et al., 2003; Salgado et al., 2003).”
• The changing epidemiology of resistance. (2009, background): “Panton–Valentine leukocidin genes), significantly more likely to be found than in hospital-acquired isolates.(42) These strains are genetically unrelated to hospital-acquired strains of MRSA and in some centres have emerged as the predominant cause of SSTIs, with the USA300 clone being the most freq”
• Community-Associated Methicillin-Resistant Staphylococcus aureus: Epidemiology and Clinical Consequences of an Emerging Epidemic (2010, background): “PVL , but only 4% of HA-MRSA isolates were PVL (654).”
• Prevalence of Staphylococcus aureus nasal colonization in the United States, 2001-2002. (2006, background): “Higher rates of community-associated MRSA infections previously have been associated with strains characterized by susceptibility to non–b-lactam agents (with the exception of erythromycin), presence of enterotoxin H and PVL genes, and predilection of infection of persons of certain nonwhite races/e”
• Emergence of Community-Acquired Methicillin-Resistant Staphylococcus aureus USA 300 Clone as the Predominant Cause of Skin and Soft-Tissue Infections (2006, background): “infections who were hospitalized within the previous year as having health care–associated MRSA (1).”
• Methicillin-Resistant Staphylococcus aureus: Molecular Characterization, Evolution, and Epidemiology (2018, background): “As discussed above, while PVL is universally linked epidemiologically to CA-MRSA strains, it is not known how it contributes to the fitness, virulence, and pathogenicity of these strains or if it is merely a marker for other fitness or virulence determinants (610).”
• The relationship between antimicrobial resistance and patient outcomes: mortality, length of hospital stay, and health care costs. (2006, background): “doubling time and the higher proportion of isolates with Panton-Valentine leukocidin gene and other exotoxin genes [38, 39].”
• The Changing Microbial Epidemiology in Cystic Fibrosis (2010, background): “%) harbored CA-MRSA, while the remainder were infected with HA-MRSA.”