Oxidative stress and inflammation, a link between chronic kidney disease and cardiovascular disease.
By V. Cachofeiro, M. Goicochea, S. G. de Vinuesa, P. Oubiña, V. Lahera, J. Luño. Kidney international. Supplement, 2008
643 citations recorded by the source corpus.
Subfields: Cardiology and Cardiovascular Medicine, Nephrology
View Paper: https://www.semanticscholar.org/paper/eee5fc3ad02a8cf5bed58ef8149eb10d54986135
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Citations in the corpus
- Oxidant Mechanisms in Renal Injury and Disease (2016, positive): “Renal Sources of Oxidative Stress The role of ROS and RNS is considered a critical factor involved in initiation and progression of various mammalian kidney diseases and injuries, including diabetic nephropathy, hypertensionrelated kidney injury, ischemia-reperfusion injury, toxic-induced nephropath”
- Oxidative stress, aging, and diseases (2018, background): “Endothelial dysfunction, induced by oxidative stress, changes vascular permeability and leads to the entry of LDL cholesterol into intimal layer and increased oxLDL levels, which are involved in the atherosclerosis process, as mentioned earlier.(48) ADMA-induced endothelial dysfunction leads to prot”
- Expansion of urease- and uricase-containing, indole- and p-cresol-forming and contraction of short chain fatty acid-producing intestinal microbiota in ESRD (2014, background): “…of these products, the intestinal epithelial barrier impairment leads to local and systemic inflammation, which are invariably present in ESRD patients and play a major role in the pathogenesis of cardiovascular disease, anemia, protein energy wasting and various other complications [27–29] .”
- Role of ROS and Nutritional Antioxidants in Human Diseases (2018, background): “With the continuous propagation of SMCs, monocytes, and macrophages, fatty streaks are ultimately converted into more advanced fibrous plaque (Madamanchi et al., 2005), potentially leading to vessel occlusion (Cachofeiro et al., 2008).”