Roles for insulin receptor, PI3-kinase, and Akt in insulin-signaling pathways related to production of nitric oxide in human vascular endothelial cells.

By G. Zeng, F. Nystrom, L. V. Ravichandran, L. Cong, M. Kirby, H. Mostowski, M. Quon. Circulation, 2000

782 citations recorded by the source corpus.

Subfields: Epidemiology, Molecular Biology, Physiology

View Paper: https://www.semanticscholar.org/paper/e894e8aa3ffd725bd613557fe973b8ca1ac22471

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Citations in the corpus

• Endothelial dysfunction in diabetes mellitus (2007, neutral): “Second, insulin is a vasodilator stimulating expression of eNOS through the phosphatidylinositol 3-kinase (PI3K) pathway (Zeng et al 2000).”
• Cardiovascular actions of insulin. (2007, background): “The first study directly demonstrating that insulin stimulates production of NO from endothelial cells used an NO-selective electrode to show that levels of NO produced in human umbilical vein endothelial cells (HUVEC) in response to a maximally stimulating concentration of insulin are approximately”