Increased rectal mucosal enteroendocrine cells, T lymphocytes, and increased gut permeability following acuteCampylobacter enteritis and in post-dysenteric irritable bowel syndrome

By R. Spiller, D. Jenkins, J. Thornley, J. Hebden, T. Wright, M. Skinner, K. Neal. Gut, 2000

1,194 citations recorded by the source corpus.

Subfields: Food Science, Gastroenterology, Genetics

View Paper: https://www.semanticscholar.org/paper/e3e7dc9baddeeea7f11fad497670c691ae9acfa0

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1. Maria Saarela

Citations in the corpus

• Postinfectious irritable bowel syndrome. (2008, background): “Campylobacter infection has been shown to lead to an increase in the number of enterochromaffin cells [3], possibly as a response to mucosal injury and inflammation.”
• Analysis of the Fecal Microbiota of Irritable Bowel Syndrome Patients and Healthy Controls with Real-Time PCR (2005, result): “jejuni is in accordance with the previously reported relationship between this pathogen and IBS (4, 26); however, this is a solitary finding.”