Particulate air pollution induces progression of atherosclerosis.

By Tatsushi Suwa, J. Hogg, K. Quinlan, A. Ohgami, R. Vincent, S. V. van Eeden. Journal of the American College of Cardiology, 2002

614 citations recorded by the source corpus.

Subfields: Health, Toxicology and Mutagenesis, Immunology

View Paper: https://www.semanticscholar.org/paper/e0c0e18bc8a9cc49e2dfa4dca97bda32650fcca1

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Citations in the corpus

Potential Role of Ultrafine Particles in Associations between Airborne Particle Mass and Cardiovascular Health (2005, background): “…instillation of ambient urban PM10 led to an increase in circulating polymorphonuclear neutrophils and caused an increase in the volume fraction of atherosclerotic lesions, which correlated with the number of alveolar macrophages that phagocytosed PM10 in the lung (r = 0.5) (Suwa et al. 2002).”
Ambient Air Pollution and Atherosclerosis in Los Angeles (2004, background): “Overall, the various limitations in our exposure assignment may add some random error, biasing results toward weaker associations (Thomas et al. 1993).”
Health effects of particulate air pollution: A review of epidemiological evidence (2011, methodology): “…exposure to the level of leukocytes (Ghio et al., 2003; Ruckerl et al., 2006; Ruckerl et al., 2007b; Schwartz, 2001; Seaton et al., 1999; Suwa et al., 2002; Tan et al., 2000; van Eeden et al., 2001); however, results are contradictory and different hypotheses regarding the underlying…”
Drug delivery and nanoparticles: Applications and hazards (2008, background): “Inﬂ ammation in distal sites has long been associated with destabilization of atheromatous plaques and both instillation and inhalation of PM cause morphological evidence of atheromatous plaque increase and destabilization in rabbits (Suwa et al 2002) and mice (Chen and Nadziejko 2005).”
A review on the human health impact of airborne particulate matter. (2015, background): “Moreover, the potent role of PM in increased cardiac risk is also explained by initiating and promoting atherosclerotic progression, which is responsible for most cardiovascular diseases (Suwa et al., 2002).”
Oxidative Stress and Air Pollution Exposure (2011, background): “[74] reported that exposure to PM10 cause progression of atherosclerotic lesions towards a more advanced phenotype hyperlipidemic rabbits.”
Ambient Particulate Pollutants in the Ultrafine Range Promote Early Atherosclerosis and Systemic Oxidative Stress (2008, methodology): “Although the methodology used is sensitive and specific for the determination of MDA,28 there are several limitations in this assay in reflecting the degree of lipid peroxidation, as reviewed by Janero et al,29, such as: (1) MDA yield as a result of lipid peroxidation varies with the nature of the p”
Adverse cardiovascular effects of air pollution (2009, methodology): “In the Watanabe hyperlipidemic rabbit model, repeated instillation of ambient PM 10 was associated with the development of more-advanced, 'vulnerable' coronary and aortic atherosclerotic plaques than those seen in control rabbits”
Long-term air pollution exposure and acceleration of atherosclerosis and vascular inflammation in an animal model. (2005, background): “Second, a marked systemic and pulmonary inflammatory response was noted in the rabbit study.(16) This suggests exposure to very high levels of particulate matter that may not exactly mirror the in vivo clinical context.”