Acceleration of intestinal polyposis through prostaglandin receptor EP2 in ApcΔ716 knockout mice

By M. Sonoshita, K. Takaku, N. Sasaki, Y. Sugimoto, F. Ushikubi, S. Narumiya, M. Oshima, M. Taketo. Nature Medicine, 2001

598 citations recorded by the source corpus.

Subfields: Pharmacology, Small Animals, Surgery

View Paper: https://www.semanticscholar.org/paper/e084d38d8f8844ae6d3cbe8c53807e6ac2c7e5e6

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Citations in the corpus

• The COX-2/PGE2 pathway: key roles in the hallmarks of cancer and adaptation to the tumour microenvironment. (2009, background): “reduction in PGE2, since the intestinal adenomas present in APC mice that are null for the PGE2 receptor EP2 are defective in VEGF induction [101].”
• At the crossroads of inflammation and cancer. (2004, unknown): “EP1 or EP3 (Sonoshita et al., 2001).”
• The Role of COX-2 in Intestinal Inflammation and Colorectal Cancer (2009, unknown): “The central role of PGE2 in colorectal tumorigenesis has been further confirmed by evaluating mice with homozygous deletion of PGE2 receptors (Watanabe et al., 1999; Sonoshita et al., 2001; Mutoh et al., 2002).”